许店文｜甘肃农业大学｜学者主页

2025

Correction to "Stigmasterol Activates the mTOR Signaling Pathway by Inhibiting ORP5 Ubiquitination to Promote Milk Synthesis in Bovine Mammary Epithelial Cells".

Ming-Yang Sun, Yu Cao, Ji Cheng, Dianwen Xu, Feng Li, Jiaxin Wang, Yusong Ge, Yuhao Liu, Xiaoyu Long, Wen-Jin Guo, Ju-Xiong Liu, Shou-Peng Fu

Journal of Agricultural and Food Chemistry

DOI

2025

Stigmasterol from Prunella vulgaris L. Alleviates LPS-induced mammary gland injury by inhibiting inflammation and ferroptosis.

Ming-Yang Sun, Dianwen Xu, Dian-Feng Liu, X. Ran, Feng Li, Jiaxin Wang, Yusong Ge, Yuhao Liu, Wen-Jin Guo, Ju-Xiong Liu, Yu Cao, Shou-Peng Fu

Phytomedicine

DOI

2024

线粒体相关内质网膜调节线粒体自噬在金黄色葡萄球菌乳腺炎中的作用及机制研究

许店文

《吉林大学》

2024

Stigmasterol Activates the mTOR Signaling Pathway by Inhibiting ORP5 Ubiquitination to Promote Milk Synthesis in Bovine Mammary Epithelial Cells.

Ming-Yang Sun, Yu Cao, Ji Cheng, Dianwen Xu, Feng Li, Jiaxin Wang, Yusong Ge, Yuhao Liu, Xiaoyu Long, Wen-Jin Guo, Ju-Xiong Liu, Shou-Peng Fu

Journal of Agricultural and Food Chemistry

DOI

2024

Hordenine Alleviates Lipopolysaccharide-Induced Mastitis by Suppressing Inflammation and Oxidative Stress, Modulating Intestinal Microbiota, and Preserving the Blood-Milk Barrier.

Yachun Xie, Xin-Yi Li, Dianwen Xu, De-Wei He, Jiaxin Wang, Jun-Long Bi, Ju-Xiong Liu, Shou-Peng Fu

Journal of Agricultural and Food Chemistry

DOI

2023

Staphylococcus aureus induces mitophagy to promote its survival within bovine mammary epithelial cells.

Dianwen Xu, Gui-Qiu Hu, Jianchun Luo, Ji Cheng, Di Wu, Lisha Cheng, Xuejie Huang, Shou-Peng Fu, Ju-Xiong Liu

Veterinary Microbiology

DOI

2022

CIDEA Regulates De Novo Fatty Acid Synthesis in Bovine Mammary Epithelial Cells by Targeting the AMPK/PPARγ Axis and Regulating SREBP1.

Ji Cheng, Dianwen Xu, Lisha Chen, Wenjin Guo, Gui-Qiu Hu, Ju-Xiong Liu, Shou-Peng Fu

Journal of Agricultural and Food Chemistry

DOI

2021

五味子甲素对LPS诱导的小鼠乳房炎的影响及其机制

许店文

《吉林大学》

2021

Protective effect of myricetin on LPS-induced mastitis in mice through ERK1/2 and p38 protein author

Xing-Chi Kan, Ju-Xiong Liu, Yingsheng Chen, Wenjin Guo, Dianwen Xu, Ji Cheng, Yu Cao, Zhanqing Yang, Shou-Peng Fu

Naunyn-Schmiedeberg's Archives of Pharmacology

DOI

2020

Myricetin protects against H2O2‐induced oxidative damage and apoptosis in bovine mammary epithelial cells

Xing-Chi Kan, Ju-Xiong Liu, Yingsheng Chen, Wenjin Guo, Dianwen Xu, Ji Cheng, Yu Cao, Zhanqing Yang, Shou-Peng Fu

Journal of Cellular Physiology

DOI

2019

Schisandrin A protects against lipopolysaccharide-induced mastitis through activating Nrf2 signaling pathway and inducing autophagy.

Dianwen Xu, Ju-Xiong Liu, He Ma, Wenjin Guo, Jiaxin Wang, Xing-Chi Kan, Yanwei Li, Qian Gong, Yu Cao, Ji Cheng, Shou-Peng Fu

International Immunopharmacology

DOI

2019

Myricetin relieves LPS‐induced mastitis by inhibiting inflammatory response and repairing the blood–milk barrier

Xing-Chi Kan, Bingrun Liu, Wenjin Guo, Libin Wei, Yuanqing Lin, Yingcheng Guo, Qian Gong, Yanwei Li, Dianwen Xu, Yu Cao, Bing-Xu Huang, A. Dong, He Ma, Shou-Peng Fu, Ju-Xiong Liu

Journal of Cellular Physiology

DOI

2019

Vanillin protects the blood–milk barrier and inhibits the inflammatory response in LPS‐induced mastitis in mice

Wenjin Guo, Bingrun Liu, Gui-Qiu Hu, Xing-Chi Kan, Yanwei Li, Qian Gong, Dianwen Xu, He Ma, Yu Cao, Bing-Xu Huang, Shou-Peng Fu, Ju-Xiong Liu

Toxicology and Applied Pharmacology

DOI

2019

Licochalcone A Protects the Blood–Milk Barrier Integrity and Relieves the Inflammatory Response in LPS-Induced Mastitis

Wenjin Guo, Bingrun Liu, Yun-Hou Yin, Xing-Chi Kan, Qian Gong, Yanwei Li, Yu Cao, Jian-Fa Wang, Dianwen Xu, He Ma, Shou-Peng Fu, Ju-Xiong Liu

Frontiers in Immunology

DOI

2018

Peiminine Protects against Lipopolysaccharide-Induced Mastitis by Inhibiting the AKT/NF-κB, ERK1/2 and p38 Signaling Pathways

Qian Gong, Yanwei Li, He Ma, Wenjin Guo, Xing-Chi Kan, Dianwen Xu, Ju-Xiong Liu, Shou-Peng Fu

International Journal of Molecular Sciences

DOI

2018

Farrerol Relieve Lipopolysaccharide (LPS)-Induced Mastitis by Inhibiting AKT/NF-κB p65, ERK1/2 and P38 Signaling Pathway

Yanwei Li, Qian Gong, Wenjin Guo, Xing-Chi Kan, Dianwen Xu, He Ma, Shou-Peng Fu, Ju-Xiong Liu

International Journal of Molecular Sciences

DOI

许店文

精选作品

期刊论文

Correction to "Stigmasterol Activates the mTOR Signaling Pathway by Inhibiting ORP5 Ubiquitination to Promote Milk Synthesis in Bovine Mammary Epithelial Cells".

Stigmasterol from Prunella vulgaris L. Alleviates LPS-induced mammary gland injury by inhibiting inflammation and ferroptosis.

线粒体相关内质网膜调节线粒体自噬在金黄色葡萄球菌乳腺炎中的作用及机制研究

Stigmasterol Activates the mTOR Signaling Pathway by Inhibiting ORP5 Ubiquitination to Promote Milk Synthesis in Bovine Mammary Epithelial Cells.

Hordenine Alleviates Lipopolysaccharide-Induced Mastitis by Suppressing Inflammation and Oxidative Stress, Modulating Intestinal Microbiota, and Preserving the Blood-Milk Barrier.

Staphylococcus aureus induces mitophagy to promote its survival within bovine mammary epithelial cells.

CIDEA Regulates De Novo Fatty Acid Synthesis in Bovine Mammary Epithelial Cells by Targeting the AMPK/PPARγ Axis and Regulating SREBP1.

五味子甲素对LPS诱导的小鼠乳房炎的影响及其机制

Protective effect of myricetin on LPS-induced mastitis in mice through ERK1/2 and p38 protein author

Myricetin protects against H2O2‐induced oxidative damage and apoptosis in bovine mammary epithelial cells

Schisandrin A protects against lipopolysaccharide-induced mastitis through activating Nrf2 signaling pathway and inducing autophagy.

Myricetin relieves LPS‐induced mastitis by inhibiting inflammatory response and repairing the blood–milk barrier

Vanillin protects the blood–milk barrier and inhibits the inflammatory response in LPS‐induced mastitis in mice

Licochalcone A Protects the Blood–Milk Barrier Integrity and Relieves the Inflammatory Response in LPS-Induced Mastitis

Peiminine Protects against Lipopolysaccharide-Induced Mastitis by Inhibiting the AKT/NF-κB, ERK1/2 and p38 Signaling Pathways

Farrerol Relieve Lipopolysaccharide (LPS)-Induced Mastitis by Inhibiting AKT/NF-κB p65, ERK1/2 and P38 Signaling Pathway

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